Mycosporine - like amino acids ( MAAs ) strongly absorb UVR and protect cells against harmful UVR.
類菌孢素氨基酸(mycosporine-like aminoacids,間稱MAAs ) 存在于許多生物體內,對紫外輻射 ( UVR) 有較高的吸收能力,被認為有保護細胞、減少UVR損傷的功能.
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Objective To investigate the protective effect of nicotinamide on UVR - induced DNA damage.
目的探討煙酰胺對紫外線照射誘發DNA損傷的保護作用.
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Thus, risk evaluation of cataract induced by UVR is an urgent need.
因此, 亟待開展紫外線與白內障的危險性評價.
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There was a 9.97 % ~67.1 % decrease in the population of LC after UVR.
表皮內LC密度降低,細胞數較照射前減少9.7%~67.01%.
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The density and structure of epidermal LC returned to normal 8 days after cessation of UVR.
停止照射后8天LC密度及結構基本復原.
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Conclusion nicotinamide is a perfect antagonist to UVR - induced DNA damage within fitting concentration.
結論適宜濃度的煙酰胺是抗紫外線照射誘發DNA損傷的拮 抗 劑.
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UVR and chemicals are two major regulating factors which affecting the melanin genesis or melanin transport.
雖然影響體內黑素合成及運輸的因素繁多,黑素合成機制也較為復雜,但紫外線和化學物質是影響和調控膚色的兩大主要因素.
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Objective To investigate the relationship between personal exposure of ultraviolet radiation and ambient solar UVR.
目的 了解個體紫外線暴露劑量與環境紫外線劑量的關系.
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