ATRA

Curcumin , a JNK blocker , blocked ATRA inhibition of cell growth.

atra對 Y79 細胞的抑制作用可被JNK的 阻斷劑Curcumin阻斷.

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The TM transcription and expression upregulated by ATRA may alleviate dysfunction of coagulation in APL.

ATRA增高TM轉錄及表達,緩解APL患者凝血功能異常.

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Curcumin , a JNK blocker, blocked the apoptosis and the growth inhibition induced by ATRA.

ATRA可誘導Y79細胞凋亡,此凋亡過程可被JNK的 阻斷劑Curcumin阻斷.

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Objective : To detect the mechanism of hyperleukocytosis in patients with APL after treatment with ATRA.

目的: 揭示APL患者ATRA治療后高白細胞征的機制.

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IntroductionAll - trans retinoic acid ( ATRA ) is efficiently differentiation in - ducer for acute promyelocytic leukemia ( APL ).

全反式維甲酸 ( ATRA ) 用于急性早幼粒細胞白血病 ( APL) 的誘導分化治療,有很高的完全緩解(CR ) 率,現已成為APL誘導緩解治療的首選藥物.

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Curcumin, JNK blocker , blocked the growth inhibition by ATRA.

此生長抑制過程可被 JNK 的阻斷劑Curcumin阻斷.

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Conclusion There are significant improvement in prognosis of APL treated with ATRA.

結論全反式維甲酸對急性早幼粒細胞性白血病患者的預后有很大改善.

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Objective To observe the clinical effects of all - trans - retinoic acid ( ATRA ) in treating 11 patients with acute promyelocytic leukemia ( APL ).

目的觀察11例急性早幼粒細胞性白血病患者經全反式 維 甲酸 治療后的臨床效果.

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Objective To determine the effect of all - trans retinoic acid ( ATRA ) on human tongue carcinoma.

目的研究維 甲酸 對舌癌細胞株的誘導分化作用.

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In contrast, APL cells of two cases showed synergistic differentiation after treatment with ATAR and bufalin.

為了進一步研究蟾蜍靈對ATRA誘導分化的增強效應, 我們研究蟾蜍靈與維甲酸聯合對APL原 代培養細胞的誘導分化作用.

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